Current foci here includes preservation of infant-transferase systems and maintenance for compromised, post-infantile systems.

Suggestions [data] presently include injury to transferase systems incurred in some circumstances through introduction of substrates via routine administrations. (Layman's Terms: At minimum, any pre-born child with a birthed sibling should benefit from observations into sibling DNA for risk factors before receiving regular shots. Adolescents and adults with any health concerns mental or physical should test and consult prior to immunizations.)

MAYO CLINIC, Abstract: "Background: The objective was to assess the association between increasing autism incidence rates and the increasing dose of folic acid in prescription prenatal and pediatric vitamins. Methods: We used published autism incidence rates from the Rochester Epidemiological Project in Rochester, MN, for 1976-1997. Additionally, we used the percent of prescription prenatal vitamins containing [...] Conclusions: If it is true that too little folic acid results in nervous tissue damage, as is accepted by the scientific community in regard to neural tube defects (NTDs), then it seems plausible that too much folic acid may result in nervous tissue damage associated with autism. Although the correlations described here do not provide proof of causation, these data provide an impetus for further study. Children who develop autism may be receiving a massive dose of folic acid in utero, as well as, after birth. It would be of interest to carry out a case-control study using medical record data to document folic acid intake for pregnant women whose offspring were later diagnosed with autism and controls." [See Methyl Health Case Studies further below.]